Acute inflammation - Neutrophils
Chronic inflammation - macrophages, lymphocytes and plasma cells
What occurs
Fluid movement across capillaries - Starlings Hypothesis
However in inflammation:
increased hydrostatic pressure → damage to endothelial cells → increased protein concentration in blood
Mediators of inflammatory response: Plasma proteins & mediators from cells (cytokines, histamine)
Anti-inflammatory drugs target these mediators
Local accumulation of neutrophils = pus
within hollow viscus → empyema
within solid organ → abscess
Effects of acute inflammation